| Correcting for ascertainment bias in the COGA data set |
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| Power loss for multiallelic transmission/disequilibrium test when errors introduced: GAW11 simulated data |
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| Nonparametric linkage and family‐based association studies of a simulated complex disorder |
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| Application of probabilistic neural network analysis to a disease with complex inheritance: The GAW11 simulated data |
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| The impact of marker allele frequency misspecification in variance components quantitative trait locus analysis using sibship data |
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| Assessment of estimation procedures for risk and onset hazard with dependent data |
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| Detection of quantitative trait loci associated with alcohol‐dependence: Use of model‐free sib‐pair method and combined segregation‐linkage analysis based on regressive models |
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| Linkage and association analyses of alcoholism using a regression‐based transmission/disequilibrium test |
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| Model‐based and model‐free multipoint genome‐wide linkage analysis of alcoholism |
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| A semiquantitative whole genome screen analysis of alcohol dependence |
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| Alcoholism as a complex trait: Comparison of genetic models and role of epidemiological risk factors |
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✓ |
✓ |
ethnicity |
| Linkage of chromosome 1 markers to alcoholism‐related phenotypes by sib pair linkage analysis of principal components |
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| Genome scans for genetic predisposition to alcoholism by use of transmission disequilibrium test analyses |
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✓ |
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| Genome‐wide linkage analysis using genetic variance components of alcohol dependency‐associated censored and continuous traits |
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| Analyses of the COGA data set in one ethnic group with examinations of alternative definitions of alcoholism |
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✓ |
✓ |
single ethnic group |
| Effects of genotype×sex interaction on linkage analysis of visual event‐related evoked potentials |
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| Evidence for linkage and association to alcohol dependence on chromosome 19 |
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| A more powerful method to evaluate p‐values in GENEHUNTER |
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| Simulated data for a complex genetic trait (Problem 2 for GAW11): How the model was developed, and why |
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| Genetic model‐free linkage analysis using the maximum‐likelihood‐binomial method for categorical traits |
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| Analysis of complex traits using neural networks |
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| Multipoint linkage analysis using the weighted‐pairwise correlation statistic |
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| Analysis of simulated data: Evidence for genetic and environmental effects |
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| Hunting genetic diseases: Exploring a multistage approach to identifying disease loci |
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| A joint test of linkage and gene×environment interaction, with affected sib pairs |
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| Impact of family structure on the power of linkage tests using sib‐pair methods |
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| Validation of linkage by sampling based on environmental exposures |
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| Masthead |
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| Preface |
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| Exploring genetic analysis of complex traits through the paradigm of alcohol dependence: Summary of GAW11 contributions |
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| Mapping alcoholism genes using linkage/linkage disequilibrium analysis |
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| A genome‐wide scan for a simulated data set using two newly developed methods |
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| Environmental covariates: Effects on the power of sib‐pair linkage methods |
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| The effect of phenotype variation on detection of linkage in the COGA data |
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| An evaluation of affected‐sib‐pair methods and transmission/disequilibrium tests for detecting genes underlying a complex trait |
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| Oligogenic model selection using the bayesian information criterion: Linkage analysis of the P300 Cz event‐related brain potential |
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| Stratification techniques to explore genotype environment interactions |
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| Significant evidence for linkage of a simulated trait to D1G024–A conclusion reached using multiallelic transmission/disequilibrium tests |
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| Cleaning genotype data |
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| A low density genome‐wide search for loci involved in alcohol dependence using the transmission/disequilibrium test, sib‐tdt, and two combined tests |
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| Genetic analysis of personality traits and alcoholism using a mixed discrete continuous trait variance component model |
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| Exploring the impact of extended phenotype in stratified samples |
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| A multiple locus analysis of the collaborative study on the genetics of alcoholism data set |
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| Exploring linkage for alcoholism using affection status and quantitative event related potential phenotypes |
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| Evidence of linkage in subtypes of alcoholism |
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| Sex‐based linkage analysis of alcoholism |
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| A genome‐wide search for susceptibility genes linked to alcohol dependence |
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| Analysis of principal component based quantitative phenotypes for alcoholism |
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| Family density of alcoholism and linkage information in the analysis of the COGA data |
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| Genetic linkage analysis of simulated complex disease data |
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| Complete genomic screen for disease susceptibility loci in nuclear families |
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| Strategies for detecting susceptibility genes in a complex disease |
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| A simple allele sharing statistic for multiple locus systems |
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| Evaluation of the contribution of environmental factors |
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| Search for susceptibility genes, gene×gene interactions, and gene×environment interactions utilizing nonparametric linkage analysis |
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| Detection and modeling of disease susceptibility locus effects: How much can be learned from contrast of populations? |
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| Meta‐analysis of genetic linkage to quantitative trait loci with study‐specific covariates: A mixed‐effects model |
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| Acknowledgments |
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| Description of the genetic analysis workshop 11 collaborative study on the genetics of alcoholism |
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| Brain event‐related potentials, dopamine D2 receptor gene polymorphism, and smoking |
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| A genome‐wide search for loci contributing to smoking and alcoholism |
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| Markov chain monte carlo linkage analysis of a complex qualitative phenotype |
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| Genetic dissection of a complex trait |
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| A comparison of some allele‐sharing based linkage analysis methods for detecting complex trait loci |
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| A comparison of two algorithms, multimap and gene mapping system, for automated construction of genetic linkage maps |
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| Departure from the triangle constraints in discordant sib pairs: A test for genetic heterogeneity |
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| Mapping genotype to phenotype for linkage analysis |
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| Systematic search for susceptibility genes in different populations |
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| Exploring the role of environmental factors in association and linkage studies |
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| Detecting interactions between gene, site, and environmental variables using GAP |
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| A bayesian markov chain monte carlo approach to map disease genes in simulated GAW11 data |
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| Modeling linkage and association with evaluation of common sampling schemes |
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| Using case‐control designs for genome‐wide screening for associations between genetic markers and disease susceptibility loci |
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| Comparison of GENEHUNTER and MFLINK for analysis of COGA linkage data |
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| Nonparametric linkage analysis of alcohol dependence with chromosome 1 and 7 markers |
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| Smoking behavior is under the influence of a major quantitative trait locus on human chromosome 5q |
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| The impact of redefining affection status for alcoholism on affected‐sib‐pair analysis |
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| Familial analysis of event related potentials |
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| Sib‐pair linkage analyses of alcoholism: Dichotomous and quantitative measures |
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| Incorporating larger families in identity‐by‐descent based linkage analysis |
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| Novel tests for marker‐disease association using the collaborative study on the genetics of alcoholism data |
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| Haplotype analysis in the collaborative study on the genetics of alcoholism data: Double recombinants |
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| Association and linkage analysis of ICD‐10 diagnosis for alcoholism |
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| A logistic regression extension of the transmission disequilibrium test for continuous traits: Application to linkage disequilibrium between alcoholism and the candidate genes <i>DRD2</i> and <i>ADH3< |
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| Asymptotic power of likelihood‐ratio tests for detecting quantitative trait loci using the COGA data |
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| Genome search for alcohol dependence using the weighted pairwise correlation linkage method: Interesting findings on chromosome 4 |
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✓ |
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✓ |
✓ |
White, Hispanic; White, non-Hispanic; Hispanic |
| Disease‐marker associations: Power and heterogeneity in independent population samples |
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| Improving the power for disease locus detection in affected‐sib‐pair studies by using two‐locus analysis and multiple regression methods |
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| Performance of the nonparametric linkage analysis using GENEHUNTER for a complicated genetic disease |
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| A method for meta‐analysis of genome searches: Application to simulated data |
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| Evaluation of replication studies, combined data analysis, and analytical methods in complex diseases |
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| Comparison of empirical strategies to maximize GENEHUNTER lod scores |
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| Monte carlo markov chain methods for genome screening |
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| Comparison of two linkage inference procedures for genes related to the P300 component of the event related potential |
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| An empirical test of the significance of an observed quantitative trait locus effect that preserves additive genetic variation |
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| Sib‐pair analysis of the collaborative study on the genetics of alcoholism data set |
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| Searching for alcoholism susceptibility genes using markov chain monte carlo methods |
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| Identifying influential individuals in linkage analysis: Application to a quantitative trait locus detected in the COGA data |
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| Structural decomposition of genetic diversity in families with alcoholism |
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| Whole genome association studies for genes affecting alcohol dependence |
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| Application of an ordered subset analysis approach to the genetics of alcoholism |
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| Parental sex effect in families with alcoholism |
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| Summary of analyses of problem 2 simulated data for GAW11 |
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| Genetic analysis of a complex disease in the presence of an environmental risk factor |
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| Meta‐analysis by combining parameter estimates: Simulated linkage studies |
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| Assessing linkage of monoamine oxidase B in a genome‐wide scan using a univariate variance components approach |
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✓ |
✓ |
ethnicity |
| A normalized identity‐by‐state statistic for linkage analysis of sib pairs |
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| Association tests using unaffected‐sibling versus pseudo‐sibling controls |
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| A generalized estimating equations approach to linkage analysis in sibships in relation to multiple markers and exposure factors |
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| Possible linkage of alcoholism, monoamine oxidase activity and P300 amplitude to markers on chromosome 12q24 |
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| Design of artificial neural network and its applications to the analysis of alcoholism data |
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| Quantitative trait transmission disequilibrium test: Allowance for missing parents |
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| Linkage analysis with adequate modeling of a parent‐of‐origin effect |
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| Sib‐pair linkage analysis of alcohol dependence taking into account covariates and age‐of‐onset variability: Evaluation of the residual approach |
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| The importance of watching our weights: How the choice of weights for non‐independent sib pairs can dramatically alter results |
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| Meta‐analysis of linkage studies |
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| Systematic search for disease loci for complex genetic traits: A study based on simulated population data |
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| Meta‐analysis by combining p‐values: Simulated linkage studies |
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| Quantitative trait locus detection using combined linkage/disequilibrium analysis |
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| Two tests of association for a susceptibility locus for families of variable size: An example using two sampling strategies |
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| Generalization of the extended transmission disequilibrium test to two unlinked disease loci |
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| Power of concordant versus discordant sib pairs at different penetrance levels |
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| Covariates in linkage analysis |
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| Issues in genomic screening: Critical values, sample sizes, and the ability to detect linkage |
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| A bayesian approach to replication of linkage findings |
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| Comparison of evidence supporting a chromosome 6 alcoholism gene |
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| Using recursive partitioning for exploration and follow‐up of linkage and association analyses |
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| Linkage analysis in alcohol dependence |
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✓ |
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| Search for a gene×environment interaction: G×E hunt |
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| Genetic determinants of variation in gallbladder disease in the Mexican-American population |
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| Roger R. Williams, M.D. (1944-1998): Cardiovascular geneticist, physician, and gentle friend |
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| Genetic and environmental contributions to size, color, shape, and other characteristics of melanocytic naevi in a sample of adolescent twins |
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| Major genetic effects on airway-parenchymal dysanapsis of the lung: The Humboldt family study |
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| Trends in prenatal diagnosis of Down syndrome and other autosomal trisomies in Scotland 1990 to 1994, with associated cytogenetic and epidemiological findings |
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| Effects of misclassification on estimates of relative risk in family history studies |
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| A genealogical study of Alzheimer disease in the Saguenay region of Quebec |
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| Lung cancer risk in families of nonsmoking probands: Heterogeneity by age at diagnosis |
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| Familial aggregation of breast cancer with early onset lung cancer |
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| Inclusion of risk factor covariates in a segregation analysis of a population-based sample of 426 breast cancer families |
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| Familial aggregation of body mass index and subcutaneous fat measures in the longitudinal Qu�bec family study |
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| Inheritance of LDL peak particle diameter: Results from a segregation analysis in Israeli families |
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| Linkage analysis of candidate obesity genes among the Mexican-American population of Starr County, Texas |
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| Maximum-likelihood-binomial method for genetic model-free linkage analysis of quantitative traits in sibships |
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| No common major gene for apolipoprotein A-I and HDL3-C levels: Evidence from bivariate segregation analysis |
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| Bivariate familial correlation analysis of quantitative traits by use of estimating equations: Application to a familial analysis of the insulin resistance syndrome |
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| Family history of coronary heart disease and pre-clinical carotid artery atherosclerosis in African Americans and whites: The ARIC study |
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| Constructing meiotic maps with known error probability |
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| Segregation analysis of the specific response to allergens: A recessive major gene controls the specific IgE response to Timothy grass pollen |
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| Method for constructing confidently ordered linkage maps |
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| Validity of family history diagnosis for dementia |
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| Effect of inbreeding avoidance on Hardy-Weinberg expectations: Examples of neutral and selected loci |
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| Abstracts from the Eighth Annual Meeting of the International Genetic Epidemiology Society |
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| Designing studies to estimate the penetrance of an identified autosomal dominant mutation: Cohort, case-control, and genotyped-proband designs |
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| Comparison of variance components and sibpair-based approaches to quantitative trait linkage analysis in unselected samples |
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| Genetic association of five apolipoprotein polymorphisms with serum lipoprotein-lipid levels in African blacks |
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|
✓ |
|
African blacks |
| Bivariate genetic analysis of fasting insulin and glucose levels |
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| Genetic variance components analysis for binary phenotypes using generalized linear mixed models (GLMMs) and Gibbs sampling |
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| Regional inference with averagedPvalues increases the power to detect linkage |
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| Multipoint linkage disequilibrium mapping with particular reference to the African-American population |
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| Inclusion of risk factor covariates in a segregation analysis of a population‐based sample of 426 breast cancer families |
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| Heritability and segregation analysis of immune responses to specific malaria antigens in Papua New Guinea |
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| Accuracy of proband reported family history: The NHLBI Family Heart Study (FHS) |
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| Fieller's theorem and linkage disequilibrium mapping |
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| Introduction of the IBD information into the weighted pairwise correlation method for linkage analysis |
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| Regression diagnostics for the Class A regressive model with quantitative phenotypes |
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| Apolipoprotein A-IV-2 allele: Association of its worldwide distribution with adult persistence of lactase and speculation on its function and origin |
|
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| Major genetic effects on airway‐parenchymal dysanapsis of the lung: The Humboldt family study |
|
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|
|
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|
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| Regional inference with averaged P values increases the power to detect linkage |
|
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| Methionine synthase D919G polymorphism is a significant but modest determinant of circulating homocysteine concentrations |
|
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| Computing probabilities of homozygosity by descent |
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| Comparison of family history measures used to identify high risk of coronary heart disease |
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| Family history of coronary heart disease and pre‐clinical carotid artery atherosclerosis in African Americans and whites: The ARIC study |
|
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|
| Familial aggregation of body mass index and subcutaneous fat measures in the longitudinal Québec family study |
|
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|
|
|
|
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|
|
| Linkage analysis of candidate obesity genes among the Mexican‐American population of Starr County, Texas |
|
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✓ |
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Mexican‐American population |
| Bivariate genetic analysis of fasting insulin and glucose levels |
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| A genealogical study of Alzheimer disease in the Saguenay region of Quebec |
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| Designing studies to estimate the penetrance of an identified autosomal dominant mutation: Cohort, case‐control, and genotyped‐proband designs |
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| Effects of misclassification on estimates of relative risk in family history studies |
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| Abstracts from the Eighth Annual Meeting of the International Genetic Epidemiology Society |
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| Genetic association of five apolipoprotein polymorphisms with serum lipoprotein‐lipid levels in African blacks |
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✓ |
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African blacks |
| Genetic variance components analysis for binary phenotypes using generalized linear mixed models (GLMMs) and Gibbs sampling |
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| Comparison of variance components and sibpair‐based approaches to quantitative trait linkage analysis in unselected samples |
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| Trends in prenatal diagnosis of Down syndrome and other autosomal trisomies in Scotland 1990 to 1994, with associated cytogenetic and epidemiological findings |
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| Method for constructing confidently ordered linkage maps |
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| Maximum‐likelihood‐binomial method for genetic model‐free linkage analysis of quantitative traits in sibships |
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| Validity of family history diagnosis for dementia |
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| Lung cancer risk in families of nonsmoking probands: Heterogeneity by age at diagnosis |
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| Likelihoods andTDT for the case-parents design |
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| Constructing meiotic maps with known error probability |
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| Inheritance of LDL peak particle diameter: Results from a segregation analysis in Israeli families |
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| Increased cancer risk among relatives of nonsmoking lung cancer cases |
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| Methods to estimate genetic components of variance for quantitative traits in family studies |
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| Arthrogryposis |
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| Indication of linkage of serum IgE levels to the interleukin-4 gene and exclusion of the contribution of the (-590 C to T) interleukin-4 promoter polymorphism to IgE variation |
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| Exploring the dense mapping of a region of potential linkage in complex disease: An example in multiple sclerosis |
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| Familial aggregation of breast cancer with early onset lung cancer |
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| Likelihood-based approach to estimating twin concordance for dichotomous traits |
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| Recent progress in understanding the genetic susceptibility to osteoporosis |
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| Roger R. Williams, M.D. (1944–1998): Cardiovascular geneticist, physician, and gentle friend |
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| No common major gene for apolipoprotein A‐I and HDL3‐C levels: Evidence from bivariate segregation analysis |
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| Genetic determinants of variation in gallbladder disease in the Mexican‐American population |
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| Optimal allele‐sharing statistics for genetic mapping using affected relatives |
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| Bivariate familial correlation analysis of quantitative traits by use of estimating equations: Application to a familial analysis of the insulin resistance syndrome |
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| Impact of demographic distribution and population growth rate on haplotypic diversity linked to a disease gene and their consequences for the estimation of recombination rate: Example of a French Cana |
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✓ |
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French Canadian population |
| Segregation analysis of the specific response to allergens: A recessive major gene controls the specific IgE response to Timothy grass pollen |
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| Genetic and environmental contributions to size, color, shape, and other characteristics of melanocytic naevi in a sample of adolescent twins |
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| Effect of inbreeding avoidance on Hardy‐Weinberg expectations: Examples of neutral and selected loci |
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| Case-parents design for gene-environment interaction |
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